Hi everyone,
Does someone have infos about this homeopathis remedy?
Thanks,
Dr. M. Franzreb Corbelletti
Castellana 171 Bajo izda., 28046 Madrid
www.drmarcofranzreb.com
Tel.: 914491957
Fax: 914491965
nitrobenzolum
-
Lynn Cremona
- Posts: 633
- Joined: Thu Apr 22, 2004 10:00 pm
Re: nitrobenzolum
Hello Marco,
you will find
Benzinum Nitricum (artificial oil of Bitter Almonds) in Clarke's Dictionary of Pract. MM
NCH website (MacRep Repertorization)
Benzinum Nitricum
http://www.nationalcenterforhomeopathy. ... m-nitricum
Open Homeopathy
http://www.openhomeo.org/openhomeopath/ ... b.&lang=en
Nitrobenzolum
82 rubrics grade 1
Best,
Lynn
-------------------------
________________________________
--
Imagine Peace
http://www.homeopathicsolutions.blogspot.com/
you will find
Benzinum Nitricum (artificial oil of Bitter Almonds) in Clarke's Dictionary of Pract. MM
NCH website (MacRep Repertorization)
Benzinum Nitricum
http://www.nationalcenterforhomeopathy. ... m-nitricum
Open Homeopathy
http://www.openhomeo.org/openhomeopath/ ... b.&lang=en
Nitrobenzolum
82 rubrics grade 1
Best,
Lynn
-------------------------
________________________________
--
Imagine Peace
http://www.homeopathicsolutions.blogspot.com/
Re: nitrobenzolum
Marco,
Here's a case from George Guess (Links, 1992), courtesy of Reference Works:
A Case of Acute Pulmonary Odema and Anoxic Encephalopathy
George Guess, M.D.
516, Merrimon Ave. Asherville, NC 2884, USA
A 67 year-old male, while out camping, developed acute dyspnoa, orthopnoa, cyanosis, and diaphoresis. He went into ventricular fibrillation in the ambulance. Cardiopulmonary resuscitation and defibrillation led to conversion to normal sinus rhythm. However he remained comatose, with severely depressed respiration. He was admitted to the coronary care unit and put on a respirator. He had a past history of two previous episodes of acute pulmonary odema.
Medical Status
BP 110/70, T 100.8 o F (38.2oC) rectal. Pupils were unresponsive to light; no spontaneous motion of limbs; no pathological reflexes. Coarse rales in chest. Regular cardiac rhythm, without murmur. EKG showed Left Bundle Branch Block with ectopic atrial rhythm. Two days after admission EKG showed no QRS, ST or T wave changes to suggest myocardial infarction. Chest X-Ray: bilateral pulmonary odema, mild cardiac enlargement consistent with congestive heart failure. By 9 days after admission chest X-Ray showed almost complete resolution of odema. Arterial blood gases during admission showed lowering of pO2 despite 100% oxygen administration. Two hours after resuscitation pO2 was 53. Gradually it elevated. Elevated white cell count (17, 000).
While in the hospital he was treated initially during CPR with the usual drugs; e.g, atropine, epinephrine, NaHCO3, lidocaine, ventilator; later he received Decadron for cerebral odema prophylaxis; a bout of supra-ventricular tachycardia was treated with digoxin and quinidine. Diuretics were prescribed for the pulmonary odema.
Echocardiogram: sclerotic, nonstenotic aortic valve, calcified mitral annulus, minimal pericardial effusion; moderately reduced left ventricular function; moderate mitral regurgitation and mild tricuspid regurgitation.
Diagnostic impression: Global cardiac hypokinesis due to severe triple vessel coronary heart disease, causing acute pulmonary edema and ventricular fibrillation. Anoxic encephalopathy; severe respiratory acidosis.
This patient remained comatosed for 8 days despite treatment and improved vital organ functioning, though pulmonary odema was still present. I was called on the 8th day and asked if I might help. He was still on the respirator. With the aid of an astute observer at the scene in the coronary care unit the following information was obtained: comatosed and unresponsive; cold to touch; eyes open but vacant and unresponsive; slow side-to-side constant nystagmus; periodic jerking of head and head tremor; lies with head and neck hyperextended. No other unusual data was available.
Prescription:: Benzinum nitricum 30C, three times a day, as needed.
[Kent's Repertory: Eye, motions, pendulum-like, from side to side: Agar, amyg, Ars, benz-n, Carb-h, cic, Cupr, Gels, sabad, sulph]
Clarke's description of Benzinum nitricum reads:
"It produces faintness, sinking...stupor. It slows the respiration until death occurs. Rolling of the eyeballs in their vertical axis is a prominent feature, and the pupils are dilated... Head drawn backward and to the left side in spasms....Paralysis of all the limbs.... Heart: pulse rapid, weak, irregular."
The choice seemed to lie between Benzinum nitricum and Carboneum hydrogenisatum, another coma remedy noted for the oscillating motions of the eyes.
On the next day, within 24 hours, the patient very rapidly regained consciousness; all the above symptoms vanished and his vital functioning improved considerably. He was removed from the coronary care unit the following day. His physicians commented that his recovery seemed remarkably rapid.
Subsequently this patient was transfered to a custodial facility and later home. Despite normalized cardiac functioning and no localizing neurologic findings, he experienced very slowly resolving weakness of the arms and a peculiar lack of strength of speech; I am told that he still suffers from a neurologic impairment of his speech. He has moved and been unavailable for further prescribing.
His response to the homoopathic remedy was dramatic and rapid; those observers who were close had no doubt as to its efficacy. This case reveals how homoopathy, administered in concert with allopathic medications, can greatly facilitate clinical response in dire circumstances.
Peace,
Dale
________________________________
Here's a case from George Guess (Links, 1992), courtesy of Reference Works:
A Case of Acute Pulmonary Odema and Anoxic Encephalopathy
George Guess, M.D.
516, Merrimon Ave. Asherville, NC 2884, USA
A 67 year-old male, while out camping, developed acute dyspnoa, orthopnoa, cyanosis, and diaphoresis. He went into ventricular fibrillation in the ambulance. Cardiopulmonary resuscitation and defibrillation led to conversion to normal sinus rhythm. However he remained comatose, with severely depressed respiration. He was admitted to the coronary care unit and put on a respirator. He had a past history of two previous episodes of acute pulmonary odema.
Medical Status
BP 110/70, T 100.8 o F (38.2oC) rectal. Pupils were unresponsive to light; no spontaneous motion of limbs; no pathological reflexes. Coarse rales in chest. Regular cardiac rhythm, without murmur. EKG showed Left Bundle Branch Block with ectopic atrial rhythm. Two days after admission EKG showed no QRS, ST or T wave changes to suggest myocardial infarction. Chest X-Ray: bilateral pulmonary odema, mild cardiac enlargement consistent with congestive heart failure. By 9 days after admission chest X-Ray showed almost complete resolution of odema. Arterial blood gases during admission showed lowering of pO2 despite 100% oxygen administration. Two hours after resuscitation pO2 was 53. Gradually it elevated. Elevated white cell count (17, 000).
While in the hospital he was treated initially during CPR with the usual drugs; e.g, atropine, epinephrine, NaHCO3, lidocaine, ventilator; later he received Decadron for cerebral odema prophylaxis; a bout of supra-ventricular tachycardia was treated with digoxin and quinidine. Diuretics were prescribed for the pulmonary odema.
Echocardiogram: sclerotic, nonstenotic aortic valve, calcified mitral annulus, minimal pericardial effusion; moderately reduced left ventricular function; moderate mitral regurgitation and mild tricuspid regurgitation.
Diagnostic impression: Global cardiac hypokinesis due to severe triple vessel coronary heart disease, causing acute pulmonary edema and ventricular fibrillation. Anoxic encephalopathy; severe respiratory acidosis.
This patient remained comatosed for 8 days despite treatment and improved vital organ functioning, though pulmonary odema was still present. I was called on the 8th day and asked if I might help. He was still on the respirator. With the aid of an astute observer at the scene in the coronary care unit the following information was obtained: comatosed and unresponsive; cold to touch; eyes open but vacant and unresponsive; slow side-to-side constant nystagmus; periodic jerking of head and head tremor; lies with head and neck hyperextended. No other unusual data was available.
Prescription:: Benzinum nitricum 30C, three times a day, as needed.
[Kent's Repertory: Eye, motions, pendulum-like, from side to side: Agar, amyg, Ars, benz-n, Carb-h, cic, Cupr, Gels, sabad, sulph]
Clarke's description of Benzinum nitricum reads:
"It produces faintness, sinking...stupor. It slows the respiration until death occurs. Rolling of the eyeballs in their vertical axis is a prominent feature, and the pupils are dilated... Head drawn backward and to the left side in spasms....Paralysis of all the limbs.... Heart: pulse rapid, weak, irregular."
The choice seemed to lie between Benzinum nitricum and Carboneum hydrogenisatum, another coma remedy noted for the oscillating motions of the eyes.
On the next day, within 24 hours, the patient very rapidly regained consciousness; all the above symptoms vanished and his vital functioning improved considerably. He was removed from the coronary care unit the following day. His physicians commented that his recovery seemed remarkably rapid.
Subsequently this patient was transfered to a custodial facility and later home. Despite normalized cardiac functioning and no localizing neurologic findings, he experienced very slowly resolving weakness of the arms and a peculiar lack of strength of speech; I am told that he still suffers from a neurologic impairment of his speech. He has moved and been unavailable for further prescribing.
His response to the homoopathic remedy was dramatic and rapid; those observers who were close had no doubt as to its efficacy. This case reveals how homoopathy, administered in concert with allopathic medications, can greatly facilitate clinical response in dire circumstances.
Peace,
Dale
________________________________