Barbara wrote:
I've been asked by a colleague to post a request regarding a px of hers who
is an
elderly lady. The small amount of info she was able to provide was about an
embolism behind the eye, which occurred shortly after a severe headache
(subsequent to a loved one's funeral). It is her left eye. My colleague
is asking if
anyone has experience in this area, and any suggestions on how to disperse
the
blood safely, as it has left the lady blind in that eye (or maybe partially,
I'm not sure,
it was a quick discussion).
Anyone like to offer the benefit of their experiences?
-------------------------------------------------------
***no experience, however, enjoyed the research and study exercise that
your query elicited.
Following is a link to a very good read about the condition. Also, in EH as
would probably also be found in Reference Works, there is some addtional
info which may need to be taken into consideration in this case.
http://www.emedicine.com/neuro/topic681.htm#target5
It seems, doing a brief summary, that the emboli can be one of mainly 3
options, Cholesterol, Fibrin/platelet, or Calcific. Other emboli can be from
tumors, fat, microorganisms, air, and foreign matter -- however, due to the
suddeness and the lack of injury and the circumstance it is likely to be one
of the main 3 and from these most likely either fibrin/platelet or Calcific,
as cholesterol only usually causes partial obstruction which leaves some
vision.
There is increased risk of coexistent carotid or cardiac disease, and can
lead to a 10 fold increase in stroke after some time has passed following.
The problem initially is the obstruction which leads to damage to the optic
nerve. The prognosis for visual recovery is not too good if the optic nerve
then atrophies -- which happens quite easily from I read, if not treated
post haste after initial incident.
My concern, if this has been ongoing for quite awhile, would be in
preventing further vascular incidents.
Treating to cause lysis of the emboli, and then treatment of the atrophied
optic nerve would have to be decided or both employed I suspect.
If the nerve is not totally damaged, then releaving the intraocular pressure
can be aimed at.
I read that ocular massage can sometimes coax the embolus to the periphery,
and hyperbaric oxygen has been employed as well.
Here are some rubrics that seem to be relevant to consider depending on
which stage one will treat.
EYES - PHENOMENA - EMBOLISM of arteria centralis retinae (RU)
croc. OP.
EYE - EMBOLISM of arteria retina (Synth)
aml-ns. croc. op.
EYE - ATROPHY - Optic nerve (Synth)
agar. alum-p. Arg-n. ars. atox. bell. carbn-s. cina hyos. iodof. lach.
Nux-v. PHOS. PLB. santin. Stry-n. stry-p. syph. Tab. thal-xyz. Tab. verat-v.
zinc-p.
VISION - PHENOMENA - LOSS of vision, blindness - atrophy of - optic nerve,
from (RU)
syph.
EYES - PHENOMENA - ATROPHY - nerves, optic (RU)
agar. alumin-p. arg-n. ars. carbn-o. carbn-s. fil. iodof. nat-ar. NUX-V.
PHOS. santin. STRY-N. syph. TAB.
Norton proposes that for the atrophy of the optic nerve, Nux-v, Arg-nit, and
Veratr-vir. are his choice.
Has been an interesting exploration - thanks for the opportunity
Best
Robyn
PS. Read also that disease of the Spleen, kidney or liver can also lead to
production of emboli which could manifest in this way, (found in Norton I
think) and interestingly noted that in Dr. Hamers notes on Thrombus the
spleen is mentioned as well as other aspects which I would be happy to share
off list, seeing off topic on this list.
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