Hi all,
Thanks Hart for opening up this line of discussion. I hope to learn from it.
Sorry for this lengthy email. Here was my way of thinking on why or what I wrote. I look forward to you all, please, teaching me more.
Hart wrote:
See my below comments
Hart wrote:
I don't disagree. And I apologise for throwing out the term too loosely.
Hart wrote:
See my last paragraphs below.
I would say that respected Dr Mangialavori has not treated extensively in countries where acute disease is the primary form of disease treated rather than old 'encrusted" diseases born of generations of diseased states.
When practitioners from highly sophisticated & industrialized countries talk about "disease" and when practitioners from less developed countries talk about "disease", I think we're not talking about the same thing every time. That's another email, that I'll write after.
POINT:
Miasm = state of suppressed disease, or acute disease unfully cured, leading to chornic, degenerative -or in this case, congenital- disease. While the actual germ of the acute infection may not be present, the vital force has not returned to balance normalcy and still presents various symptoms.
A miasm creates a blockage to cure.
POINT:
The use of nosode in "clearing" the block to my understanding and experience, correct. In my experience, the direct nosode of the immediate "culprit" is among the very effective ways forward.
(example; one may wish to categorize the patient as "psora" "sycosis" etc, but I'd rather give the fellow some influenzinum, malaria co, typhoidinum, brucellosinum, etc instead.)
POINT:
New miasm? NO!
I not trying to name some new-fangled miasm, but rather simply saying that a miasmic state had been induced by the apparent suppression or apparent uncured whooping cough. I'm not scholarly enough to label this state "psora" or whatever.
"The proof is in the pudding" - That Pertussin acted so dramatically demonstrates to me that the hypothesis of suppressed or uncured whooping cough is validated. Generally, my practical experience in using nosodes leads me to believe that if Whooping cough wasn't a factor, then Pertussin would have had no/little effect one way or another on the child.
POINT:
On the other hand, some lines of thought in the modern thinking have named "new" aspects of miasm - eg "acute miasm", "typhoid miasm", "malaria miasm", etc. Which, by the way if you see these last two in action you'll understand very well that they create miasmic states. I'm not scholarly enough to agree or disagree or comment as to whether these suppressed diseases deserve their own special labeled miasm, but indeed miasmic they are.
Even the most common medics & nurses in Kenya observe and acknowledge that there are post-typhoidal and post-malarial states that they seem to have no idea how to treat. However when we apply homeopathic principles of miasm & suppressed disease to the medicine for these states, they clear immediately (within hours or days) and stay cleared ("cure"). I have witnessed this a number of times. Some lines of allo-thought are now pointing to cytokines as a factor. Perhaps if cytokines are factor in these miasm-like post acute states, then nosodes and tautopathics seem to be logical choices both from allopathic view as well as homeopathic view. Or perhaps better said - that now there's an allopathic concept that may help explain why nosodes - which have a kind of innoculating effect - are appropriate.
(FYI - some local nurses here nickname post-typhoid symptoms "hala-hala disease" meaning "this and that disease". Post typhoid creates nebulous wandering pains in body and joints, fatigue, unsettled stomach, etc. Often it is misdiagnosed and medicated as rhuematism.)
(FYI- post-malaria can run from 2-week periodic episodes of headaches or joint pains, chills or fever yet without any malarial parasite in the blood tests. It can also lead to convulsions or catatonic states being misdiagnosed and treated as epilepsy)
(FYI- It's still not clear to me whether these are suppressed diseases or side-effects of drugs or both.)
(ALSO I'm no expert about cytokines, only this is what I hear from discussions with local researchers.
DEFINITION Cytokine: A small protein released by cells that has a specific effect on the interactions between cells, on communications between cells or on the behavior of cells. The cytokines includes the interleukins, lymphokines and cell signal molecules, such as tumor necrosis factor and the interferons, which trigger inflammation and respond to infections.)
POINT:
It's my hypothesis that the congenital lobal emphysema was actually a state of suppressed or uncured whooping cough of recent history - ie a state that's "early chronic" or "post-acute". Tho after the operation and doctors have cleared the infant of any signs of emphysema, the infant is still prone to respiratory distress, wheezing, shortness of breath etc.
What is really still making me ponder is this question:
Hypothesis or no, then - Why / how did the whole family get sick with whooping cough and NOT the mother? Or what did really happen?
Did it pass thru her and create antibodies and no symptoms in her? Did the state of pregnancy somehow suppress or repel the disease yet could it have passed to the fetus as a suppressed state?
POINT:
I realise that there may be some readers trained and practicing in highly industrialized countries, who may not have had the chance to observe first hand "miasms in the making". Generally they've studied and are treating chronic and inherited miasms that are more common for populations in industrialized countries.
But for those who practice in SAmerica, Africa, India or parts of Asia it can still be readily observed the "live" process in action of active acute infections turning into suppressed disease states. I've frequently observed that these "post acute" miasmic states, when caught early enough, may be readily cured, they don't seem quite so complicated as the chronic cases I frequently read here on Minitus.
Sincerely,
Didi Ananda Ruchira | Director | Tels: +254 (0)733-895466 / +254 (0)723-869133 |
www.abhalight.org