Miasm physiology
Posted: Wed Jun 18, 2008 6:28 am
Dear All,
I had an interesting interchange with Dr. Leela and wanted to share it because I am hoping that there are more physiological insights into Homeopathy elsewhere. She has an article that presents miasms from a physiological perspective. I wish I had the original website for you. The title is:
Miasms in Case Management
Part 2: Disease Evolution and its Miasmatic Expression
http://www.hpathy.com/philosophy/dsouza-miasm2.asp
Here is my opening comment:
I just read your interesting article on Miasams as Physiology.
I am just a beginning physiology student and a 3rd year homeopathy student at the Toronto School of Homeopathy. Physiological interpretations help me to get a more firm grasp of the miasmatic pictures. I am wondering if immune sensitivity reactions might not also fit into this analysis. I wrote this footnote to a paper that I wrote recently, but don't expect any response. I am wondering what you think. I am just repeating what I read in my physiology textbook. I may misunderstand.
It is possible that the since the Type I hypersensitivity reactions are of the psoric type. They occur at the level of blood vessel dilation. Mostly the cells close to the skin are affected. Type II is only in some rare diseases. Type III activates the immune system, antigens and the complex cascades. Scar tissue-like obstructions are left in the tissues such as the blood vessels. Thus, entire organ systems are damaged. These are more similar to the sycotic and in more advanced stages syphilitic tissue damage. Type IV hypersensitivity reactions include tubercular reactions (Kumar 205-217). It is delayed hypersensitivity.
This physiology may apply to miasmatic divisions that you wrote about.
Thank-you,
Ellen Madono
Here's her answer:
YEs I think that is a good miasmatic interpretation of hypresensitivity.
Delayed, cell mediate hypersensitivity would be Tubercular-syphilitic depending on the expression of the individual. So there is an "active" interpretation of dominant miasmatic expression.
IF more descrtruction is prevelant in the expression, syphilitic may be predominant. If more haemorrahgic with scar tissue formation it would be tubercular.
Even Type 1 and Type 2, could have a tinge of tubercular if there is a haemorrhagic tendency in the presentation. It means that the individual has a tubercular tendency in addition to the basic miasmatic interpretation of the disease diagnosis.
Hope that doesnt' sound confusing.
nice to hear for you. Been a long time since I could get onto the List - I'm busy with both an MD (hom) and an MSc. Hope to meet you all again when I"m through with those.
warm regards,
I had an interesting interchange with Dr. Leela and wanted to share it because I am hoping that there are more physiological insights into Homeopathy elsewhere. She has an article that presents miasms from a physiological perspective. I wish I had the original website for you. The title is:
Miasms in Case Management
Part 2: Disease Evolution and its Miasmatic Expression
http://www.hpathy.com/philosophy/dsouza-miasm2.asp
Here is my opening comment:
I just read your interesting article on Miasams as Physiology.
I am just a beginning physiology student and a 3rd year homeopathy student at the Toronto School of Homeopathy. Physiological interpretations help me to get a more firm grasp of the miasmatic pictures. I am wondering if immune sensitivity reactions might not also fit into this analysis. I wrote this footnote to a paper that I wrote recently, but don't expect any response. I am wondering what you think. I am just repeating what I read in my physiology textbook. I may misunderstand.
It is possible that the since the Type I hypersensitivity reactions are of the psoric type. They occur at the level of blood vessel dilation. Mostly the cells close to the skin are affected. Type II is only in some rare diseases. Type III activates the immune system, antigens and the complex cascades. Scar tissue-like obstructions are left in the tissues such as the blood vessels. Thus, entire organ systems are damaged. These are more similar to the sycotic and in more advanced stages syphilitic tissue damage. Type IV hypersensitivity reactions include tubercular reactions (Kumar 205-217). It is delayed hypersensitivity.
This physiology may apply to miasmatic divisions that you wrote about.
Thank-you,
Ellen Madono
Here's her answer:
YEs I think that is a good miasmatic interpretation of hypresensitivity.
Delayed, cell mediate hypersensitivity would be Tubercular-syphilitic depending on the expression of the individual. So there is an "active" interpretation of dominant miasmatic expression.
IF more descrtruction is prevelant in the expression, syphilitic may be predominant. If more haemorrahgic with scar tissue formation it would be tubercular.
Even Type 1 and Type 2, could have a tinge of tubercular if there is a haemorrhagic tendency in the presentation. It means that the individual has a tubercular tendency in addition to the basic miasmatic interpretation of the disease diagnosis.
Hope that doesnt' sound confusing.
nice to hear for you. Been a long time since I could get onto the List - I'm busy with both an MD (hom) and an MSc. Hope to meet you all again when I"m through with those.
warm regards,