Hepatitis C Virus and Non-A Non-B Hepatitis
Posted: Sat Jun 07, 2008 6:03 pm
http://www.healtoronto.com/hepc.html
Is Hepatitis C Really a Virus?
Several articles here
http://www.duesberg.com/papers/ch5.html
excerpt
II. B
B. Hepatitis C Virus and Non-A Non-B Hepatitis
Non-A non-B hepatitis is observed primarily in recipients of transfusions and in intravenous drug users (3, 12, 160). It has been postulated to be a viral disease because inoculation of plasma or serum (3-75 ml) from hepatitis patients into chimpanzees induced some biochemical markers of hepatitis, such as alanine aminotransferase, in half of the animals (160). However, none of the animals developed hepatitis (161, 162). Trace amounts of presumably viral RNA have recently been detected in the liver of hepatitis patients. In addition, "nonneutralizing" antibodies to "nonstructural epitopes," from an apparently latent RNA virus, have been identified mostly in asymptomatic carriers (160). Cloning and sequencing indicated that the RNA is directly coding and measures about 10 kb. Therefore, the suspected virus has been tentatively classified as a togavirus (160). Viral RNA was only detectable after amplification with the PCR in 9 out of 15 non-A non-B hepatitis patients, and non-neutralizing antibodies were found in only 7 of the 9 RNA-positive and in 3 of the 6 RNA-negative patients (163). Likewise, liver tissues from chimpanzees inoculated with sera from hepatitis patients contain only one viral RNA molecule per ten cells (160).
In view of this evidence, the putative virus has been termed hepatitis C virus (HCV) to indicate that it is the cause of the hepatitis. Subsequently, the Food and Drug Administration has recommended, and the American Association of Blood Banks has mandated, as of 1990, the testing of the approximately 12 million annual blood donations in the U.S. (82) for antibodies to HCV at an approximate cost of $5 per test. The test was developed by Chiron Co., Emeryville, California (Irwin Memorial Blood Bank, personal communication, August 15, 1991).
However, several arguments cast doubt on the hypothesis that HCV causes hepatitis:
1. Virus-containing sera or plasma from hepatitis patients does not cause hepatitis if inoculated into chimpanzees, indicating that HCV is not sufficient to cause the disease. Moreover, since the virus has not been propagated in culture and isolated in a pure form, the possibility exists that the biochemical markers of hepatitis that are observed in chimpanzees inoculated with plasma were induced by another agent. Thus, HCV is not likely to be a sufficient cause of hepatitis in humans.
2. The presence of HCV in asymptomatic subjects at the same concentration and activity as in hepatitis patients also indicates that the virus is not sufficient to cause hepatitis.
3. The absence of viral RNA in 6 out of 15 hepatitis C patients indicates that the virus is not necessary for the disease.
It appears that HCV either causes disease by unprecedented mechanisms with as little as one RNA molecule per 10 liver cells in some and even less in other carriers, or that the virus is not the cause of non-A non-B hepatitis. By contrast, the concentration of viral RNAs made by conventional pathogenic viruses, including togaviruses, ranges from 103 to over 104 per cell (10). Therefore, it seems plausible that a latent passenger virus was identified that survives by establishing chronic asymptomatic infections at very low, nonpathogenic titers (164).
http://tinyurl.com/6gwdcl
more in book here
http://www.whale.to/a/virus.html
The Virus hunters
http://www.newmediaexplorer.org/sepp/20 ... _virus.htm
------------------------------------------
Sheri Nakken, former R.N., MA, Hahnemannian Homeopath
http://www.wellwithin1.com/vaccine.htm & http://www.wellwithin1.com/homeo.htm
ONLINE/Email classes in Homeopathy; Vaccine Dangers; Childhood Diseases Reality
Is Hepatitis C Really a Virus?
Several articles here
http://www.duesberg.com/papers/ch5.html
excerpt
II. B
B. Hepatitis C Virus and Non-A Non-B Hepatitis
Non-A non-B hepatitis is observed primarily in recipients of transfusions and in intravenous drug users (3, 12, 160). It has been postulated to be a viral disease because inoculation of plasma or serum (3-75 ml) from hepatitis patients into chimpanzees induced some biochemical markers of hepatitis, such as alanine aminotransferase, in half of the animals (160). However, none of the animals developed hepatitis (161, 162). Trace amounts of presumably viral RNA have recently been detected in the liver of hepatitis patients. In addition, "nonneutralizing" antibodies to "nonstructural epitopes," from an apparently latent RNA virus, have been identified mostly in asymptomatic carriers (160). Cloning and sequencing indicated that the RNA is directly coding and measures about 10 kb. Therefore, the suspected virus has been tentatively classified as a togavirus (160). Viral RNA was only detectable after amplification with the PCR in 9 out of 15 non-A non-B hepatitis patients, and non-neutralizing antibodies were found in only 7 of the 9 RNA-positive and in 3 of the 6 RNA-negative patients (163). Likewise, liver tissues from chimpanzees inoculated with sera from hepatitis patients contain only one viral RNA molecule per ten cells (160).
In view of this evidence, the putative virus has been termed hepatitis C virus (HCV) to indicate that it is the cause of the hepatitis. Subsequently, the Food and Drug Administration has recommended, and the American Association of Blood Banks has mandated, as of 1990, the testing of the approximately 12 million annual blood donations in the U.S. (82) for antibodies to HCV at an approximate cost of $5 per test. The test was developed by Chiron Co., Emeryville, California (Irwin Memorial Blood Bank, personal communication, August 15, 1991).
However, several arguments cast doubt on the hypothesis that HCV causes hepatitis:
1. Virus-containing sera or plasma from hepatitis patients does not cause hepatitis if inoculated into chimpanzees, indicating that HCV is not sufficient to cause the disease. Moreover, since the virus has not been propagated in culture and isolated in a pure form, the possibility exists that the biochemical markers of hepatitis that are observed in chimpanzees inoculated with plasma were induced by another agent. Thus, HCV is not likely to be a sufficient cause of hepatitis in humans.
2. The presence of HCV in asymptomatic subjects at the same concentration and activity as in hepatitis patients also indicates that the virus is not sufficient to cause hepatitis.
3. The absence of viral RNA in 6 out of 15 hepatitis C patients indicates that the virus is not necessary for the disease.
It appears that HCV either causes disease by unprecedented mechanisms with as little as one RNA molecule per 10 liver cells in some and even less in other carriers, or that the virus is not the cause of non-A non-B hepatitis. By contrast, the concentration of viral RNAs made by conventional pathogenic viruses, including togaviruses, ranges from 103 to over 104 per cell (10). Therefore, it seems plausible that a latent passenger virus was identified that survives by establishing chronic asymptomatic infections at very low, nonpathogenic titers (164).
http://tinyurl.com/6gwdcl
more in book here
http://www.whale.to/a/virus.html
The Virus hunters
http://www.newmediaexplorer.org/sepp/20 ... _virus.htm
------------------------------------------
Sheri Nakken, former R.N., MA, Hahnemannian Homeopath
http://www.wellwithin1.com/vaccine.htm & http://www.wellwithin1.com/homeo.htm
ONLINE/Email classes in Homeopathy; Vaccine Dangers; Childhood Diseases Reality