Trans Ischemic Attack?????

[Rona]

Thank you all for such great input.
I am still struggling with some of the older vocabulary such as ‘head congestion brain’ which does not specify/differentiate duration – i.e. a ‘transient event’ vs a ‘state’.

Certainly any vocabulary that indicates ‘spasm’ is easier to interpret as short term – but again – to be placed in our rep was it observed in an individual as multiple ‘spasm’ events?

For example – we would most likely never rep a single hiccup (traditional diaphragm spasm event) in a case, where we would certain rep repeated or extended duration or cyclic hiccup events, or if there were symptoms produced by these events that lingered – leaving their ‘mark’ or ‘impact’ on the individual.
Thus far(24 +hours) , she remains well – no sign or symptom produced by the event and no repeat.
Again, thank you.
Donna
From: minutus@yahoogroups.com [mailto:minutus@yahoogroups.com] On Behalf Of andyh@mcn.org
Sent: Tuesday, March 23, 2010 7:00 PM
To: minutus@yahoogroups.com
Subject: RE: [Minutus] Re: Trans Ischemic Attack?????
Hi Donna,
Robyn’s cogent opinion and the EMT guesstimate are not necessarily
mutually exclusive (as you deduced no doubt). If this is an epileptic
phenomenon, change in blood supply may still be involved, though with less
chance of organic result than in TIA or CVA from thrombosis etc:

http://emedicine.medscape.com/article/1183962-overview


Thus (Suggested by H'02)
Head Congestion Brain
is likely true even if this is more epilepsy than involving blood vessel
pathology.

An (unexpected) heart output issue like heart block (conduction) is not an
inconceivable remote cause, but maybe this is a one-time stomach-mediated
event which will lead to a proactive rx.

Her pupillary muscles did in fact go into spasm. Thus
Head Convulsion (spasm)
and
Convulsion as a general search term.

I wonder if something had happened earlier that day which could lend
additional clue to triggering cause (PT-rubbing or massage oil, etc;
someone’s comment causing anxiety (stomach); something she witnessed which
had a somatic and not conscious response).

Some rubric ideas to add:
Stomach hiccough -convulsions before – bell, CIC, cupr, hyos, ran-b, STRAM
Generalities Convulsions indigestion, from (IP, nux-v)
Head hiccough agg -bry

All of the above remedies except IP and bry make it through a cross of the
above three with
Unconsciousness, transient (though this depends on the latter being a
complete rubric)

May be useful:
Eyes, dilatation pupils convulsions during
Eyes, insensibility pupils convulsions during
Eyes movement rolling convulsions during - atro-pur, cocc

Speculative:
Eyes, dilatation pupils convulsions before? ARG-N, BUFO
Generalities, rubbing agg? (massage in PT earlier in day?)
Generalities, 5-6PM or collection of aggravation times in late afternoon
(though one instance makes it shaky premise)
Mind, Cheerful, convulsions after? (small rubric )

Best regards to you,
Andy

http://emedicine.medscape.com/article/1183962-overview


--
Robyn, Thank you - yes indeed the description does seem to fit that of a

[Shannon Nelson]

Hi Donna,

Is there a remedy that seems to cover her current overall chronic state, and is it possible she just needs a re-dose? Maybe looking for the background chronic state would be more fruitful than trying to repertorize a single fleeting event?

Best,
Shannon

[muthu kumar]

Shannon, I agree with you there -

Donna -

looks like it is the chronic state that needs to be addressed

This is a transient ischemia - symptoms have not been clear, even the hiccough might not have been a hiccough etc. THe chronic could help bring out ( hopefully) better and clear cut symptoms - we should not end up fitting the patient to the remedy -
Theoretically - you need not differentiate between transient event and a chronic state in congestion of brain or any such physical state- usually event will start a state that ends in a moment or continue - but in my experience I do not give credence to the intensity or duration of a symptom - after all our science has proved that even "sensations as if" can guide us to treating an actual symptom of the same nature... ( that is why I do not even give a lot of weightage to the various markings of distinction of symptoms... if it is present it is present) - we need to rely on the constellation of a group of a symptoms and symptom peculiarity than the intensity or duration

but in this case since the whole case at this point is dependent on something that happened very fleetingly, we need to hesitate to prescribe on it...
--- In minutus@yahoogroups.com, Shannon Nelson wrote:

[andyh]

Donna,
I hope something here helps your process with this important case.

I agree with what has been written (my paraphrase) -
that in a nervous system event as significant as sudden convulsive loss of
consciousness for 30 secs, all semiology should be at least noted, even if
this is only the "tail of an elephant". If the hiccough observation is in
question, then keep the sx questionable. Otherwise, since (for
example,Stomach hiccough -convulsions before) is in the rep with many rx,
clearly there has been a repeated observation of spasm in GI prior to or
concomitant with, or connected to convulsive phenomena elsewhere (in this
case, probably on the stomach meridian(s)). Those listed remedies bear
similarity to the manifestation. Success in removing the state which
produces the manifestation requires remedy resonance with the relevant
underlying miasmatic cause. Removing the foreign vitality creating the
pattern of defective animation which makes one susceptible to events is an
ultimate solution which motivates our finding that remedy or series of
remedies.

The chosen chronic totality which sufficiently matches the fundamental
causation as a pattern in our language of semiology may hinge on a factor
(modality, psychological picture, location, sensation, dream, etc)
*unrelated* to the observed *acute* manifestation.

However, on the other hand - within that acute symptom output may be a
discernible link to the chronic state. For example, if the chronic state
of the case had some Stram characteristic (for example used night light
inveterately as a child) then this acute event may help support that
chronic prescrip. OR - stram acute might suggest investigation of Ignatia
(according to Master, stram is the acute of Ignatia in asthma, and this
may hold in other phenomena). In the latter, the stram acute output would
suggest that not fear, but an unprocessed grief lies at the root of the
observed pathophysiology (transient shift in brain blood supply dependent
on convulsive meridian disturbance - if that is what is happening).

Or perhaps the acute sx output was determined to match bell - (and if bell
is not at all like the chronic picture) then its known chronic analogues
could be investigated (calc, lach, sil, sulph, tub).

These are just examples of how the acute manifestation may provide a clue
additive to chronic state characteristics already known or as yet queried.

The observed acute situation may or may not link in some way with the
chronic (through relationship of remedies, or being a part of the picture
of the chronic remedy at this time, or via some other connection). But if
the importance or lack thereof of the manifestation remains as yet
unknown, perhaps one of the gathered clues will become important in the
future. Data collection allows forming hypotheses to keep in mind.
Conclusion comes only after a breakthrough in understanding *which* data
hangs together as a totality which resembles a remedy in the materia
medica; relates to what needs to be cured in a case based on full
understanding of it; and addresses the largest discernible totality
possible.

If the rx of the posited totality does not give signs and lasting benefit,
then the underlying miasmatic cause is not being matched. Then other
methods may come into play (e.g. unlocking a miasm using Shahrdar's group
totality method); using a carefully selected remedy fails to act recourse
(such as anac in a case of self-possession disorder); analyzing the case
using Sankaran's method to elucidate a remedy by crossing his dominant
"miasm" with taxonomic thematics, etc... (Again, just examples of how a
case might turn..., nothing you do not already know, but I am given to
rambling a bit).

One might use data collected thus far to be prepared in event there is
another episode; so that if it is more marked in severity, some background
is in hand.

Otherwise, if nothing is amiss, no remedy should be given prior to
understanding the chronic case.

I hope this phenomenon does not recur, and you get the clues needed in
some other fashion. If it does recur, I hope it is not severe, and leads
to more answers in understanding the pathophysiology - AND pointers to a
confirmable chronic rx.

Cheers
Andy

==============
Thank you all for such great input.