The opposite is possible - the virus can copy some DNA from the cell
being invaded, and make use of it by adding it to its own viral DNA.
In fact this is the mechanism of feline leukaemia type A for example
- the virus causes leukaemia directly, but when it replicates it also
swipes part of the feline genome which when added to the virus
genome, codes for lymphoma.
The article Sheri cited claims the entire viral genome is
incorporated in the DNA and that is not possible.
What else is possible I do not know. Some discussions:
Suriya may be talking about a small piece of DNA that codes fro
something specific like a protein? I did not see what she wrote.
I do not know of such a thing.
Viral DNA is very big - 10,000 base pairs - so I can not buy that the
entire viral genome would ever be incorporated into the host cell. It
would change the function of the host cell if it did, into a mishmash
of viral activity and prior activity. So I do not buy "the viral
genome added to the DNA" as Sheri's author claims. I think it was the
author's lack of understanding of how the virus DNA lines up with and
uses cell DNA *mechanism* for making copies.
If we are looking for a miasm mechanism:
In a resistant individual the virus can NOT use the DNA to reproduce
itself.
I do not know why that is at a chemical level from any research - but
I suspect it is to do with what genes are physically accessible - how
methylated or acetylated they are and how close they are physically
to the joined aresa between two legs of a chromosome.
We DO know that genes can be switched on or off by any number of
outside influences including diet. (The mechanisms are methylation of
a gene or acetylation of a gene. The idea is that a gene can be
switched on (accessible for use) or not, by these processes. Whether
viruses can acetylate/methylate genes i do not know. Depends whether
they have the chemistry to do it?
But that would be separate from incorporating their DNA into the cell
- as in gene splicing.
I suspect methylation/acetylation of genes is how miasms get
started (rather than gene splicing) and it is also how miasms get
removed by homeopathy. Allopathic research shows diet of the
grandmother in utero - affects the chronic diseases of the
granddaughter. Same on the male side except one is beneficial change
and hte other is detrimental change. The point is that there is a
known inherited "something or other" (that allopathy has now proved
beyond doubt) and which is acquired by a prior generation (usually at
least 2 prior it seems) and passed down, and which affects chronic
diseases in those later generations.
Splicing does not make sense to me as a miasm mechanism as it does
not affect all cells including sex cells.
Methylatikon/acetylatioon does affect the body systemically so would
affect ALL cells incloouding sex cells, and thus be heritable.
Another aspect that may be relevant somewhere:
Genes are on chromosomes that look like a pair of long wiggly legs
joined somewhere along their length. At that join the legs are held
so close together that access to genes "in the crack" for use in any
way - so as to copy the DNA for a protein to make that protein (or I
surmise for access to use it for viral replication) is impossible. So
sometimes that join is strong and excludes some genes and sometimes
not. That's another way genes can be switched on/off.
This is a new area of research called epigenetics - in which one
studies how genes are switched on or off. The latest research I saw
shows how genes can be affected in one generation (being switched on
or off by outside influences especially nurtrition was mentioned) and
then the result shows up only 2 generations later, and from then
onwards, as chronic disease susceptibility.
I have not seen anything about a virus inserting DNA into the cell
DNA. This would require ability to chop out a piece (of itself??).
in addition the only cells that pass down DNA to offspring are the
sex cells. So THEY would have to be involved - not as in HIV, the
blood cells. Each virus has a type of cell they have the envelope
protein to enter. They can and do not affect all cells.
So I am having a hard time seeing a mechanism by which viral DNA can
be inserted without scrambling the cell protein activity coding, much
less getting to sex cells and being viable for combination with
another sex cell so as to be heritable.
So personally I would find a mechanism for miasms far more credible
if it had more to do with nutrition, methylation or acetylation (as
that affects ALL the cells and their gene on/off status, including
sex cells). I'd also think a methyation/acetylation trigger (to
switch on or off) could MAYBE (no evidenece or proof I know of) come
from a virus. It would be a small piece of DNA - but it would not
need to be in the genome DNA - it can be extra nuclear DNA or
mitochondrial DNA. I do not think we can prove at this point, whether
that the main chromosomal DNA is the miasm carrier:-)
We do know a virus does this:
To replicate requires aligning with the DNA to be borrowed in order
to use it like a photocopy machine to make a copy. The DNA is thus
copied (using some cell proteins to "run" the copy machine so to
speak) - but the DNA is not affected directly. So the virus
replication occurs inside the cell nucleus but does not add/'subtract
cell DNA, just copies it.
I personally have not seen research where a virus splices genes into
cell DNA.
Except fro a small specific protein I'd find it hard to even imagine.
And see no use for that except to change the local activity of the
cell so affected.
For me, the money for miasms is on epigenetics, not DNA splicing.
Namaste,
Irene
--
Irene de Villiers, B.Sc AASCA MCSSA D.I.Hom/D.Vet.Hom.
P.O. Box 4703 Spokane WA 99220.
www.angelfire.com/fl/furryboots/clickhere.html (Veterinary Homeopath.)
"Man who say it cannot be done should not interrupt one doing it."