hahnemannian2002 wrote:
I was referring to the intestines which have mucous membranes - not the
kidneys which have a different structure. There is bleeding in ONE case
of intestines just before death
The main problem with this toxin is apparently the physical crystals
that form in the kidneys, painfully....not the mucus membranes of the
gut, which is a secondary effect in some.
The key here is prolonged or massive.
But we are talking *acute* renal failure, with 40ppm.
This is not a ppm thing.
Ethylene glycol is sweet tasting to cats, it attracts them and they
drink it in volume (not in ppm) and it clogs the kidneys in a major way
- in bulk you could say.
I agree there are lots of kidney toxins. I am forever on about toxions
in pet food thatg cause kidney or other damage. even incorrect ratios of
amino acids can cause harm to kidneys. But all that takes tiem and is
dose dependant rather than being an acute case as here.
I'm hung up on the acute issue here:-))
I consider it is very relevant.
I also consider the mechanism relevant.
Ter is turpentine.
How is it similar to aminopterin?
And why does Ter not appear in the acute kidney failure rubric?
I think I know why.
IF we look at the safety data sheet for Ter in animals it says:
"Effects on Animals: Turpentine is an eye, mucous membrane, and skin
irritant and a central nervous system depressant in animals. "
...Cats exposed to a 540- to 720-ppm concentration of turpentine
exhibited signs of immediate eye and mucous membrane irritation and had
mild convulsions; at a concentration of 1440 ppm, they developed
paralysis within 150 to 180 minutes [HSDB 1989]. No adverse effects were
noted in dogs exposed to 180 ppm for 3.5 hours/day for 8 days [ACGIH
1986, p. 615]; however, raising the concentration to 818 ppm and
exposing the dogs for 3.5 to 4.5 hours caused nausea, incoordination,
mild paralysis, and weakness [Clayton and Clayton 1981, p. 3245]."
For humans it says:
"Effects on Humans: Turpentine is a skin, eye, mucous membrane, and
upper respiratory tract irritant in humans. It may also cause skin
sensitization and central nervous system, gastrointestinal, and urinary
tract effects. The lowest estimated oral dose reported to be lethal in
humans is 441 mg/kg [RTECS 1989]. Exposure to a 75-ppm concentration for
3 to 5 minutes irritates the nose and throat, and exposure to a 175-ppm
concentration irritates the eyes and may be considered intolerable by
human volunteers [Grant 1986, p. 961; Proctor, Hughes, and Fischman
1988, p. 500]. Ingestion of turpentine causes a burning pain in the
mouth and throat, nausea, vomiting, diarrhea, abdominal pain,
excitement, ataxia, confusion, stupor, seizures, fever, and tachycardia
and may cause death due to respiratory failure [Proctor, Hughes, and
Fischman 1988, p. 500]. Toxic glomerulonephritis and bladder irritation,
with hematuria, albuminuria, oliguria, and dysuria, have been associated
with overexposure to the vapor of turpentine..."
So we see some kidney effects on humans - but no acute ones. Ter is
mainly toxic in other ways. Even adding the actual proving symptoms does
not put it in the acute kidney failure category.
As a remedy with 64 kidney rubrics that specifically mentions *chronic*
kidney failure - but not acute kidney failure among the 64 - it does not
give me a lot of confidence in its PROVEN ability in acute kidney failure.
Nor does it look a good match for aminopterin to me even if I ignored
the acute kidney failure rubric (which I can not do). It has 4 of 11
best rubrics only:-)
Also - Why do you think that comparing poisons is a more valid way to
"repertorize" than the formal way of seeking appropriate rubrics to
match the case?
Namaste,
Irene
--
Irene de Villiers, B.Sc AASCA MCSSA D.I.Hom/D.Vet.Hom.
P.O. Box 4703 Spokane WA 99220.
www.angelfire.com/fl/furryboots/clickhere.html (Veterinary Homeopath.)
"Man who say it cannot be done should not interrupt one doing it."