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Our Deadly Diabetes Deception
Greed and dishonest science have promoted a lucrative worldwide epidemic of
diabetes that honesty and good science can quickly reverse by naturally
restoring the body's blood-sugar control mechanism.
Extracted from Nexus Magazine, Volume 11, Number 4 (June-July 2004)
PO Box 30, Mapleton Qld 4560 Australia. editor@nexusmagazine.com
Telephone: +61 (0)7 5442 9280; Fax: +61 (0)7 5442 9381
From our web page at: www.nexusmagazine.com by Thomas Smith © 2004
PO Box 7685
Loveland, CO 80537 USA
Email: Valley@healingmatters.com
Website: http://www.Healingmatters.com
Introduction
The Diabetes Industry
Diabetes History
Cure versus Treatment
The Commercial Value of Symptoms
Epidemiological Lifestyle Link
The Nature of the Disease
Orthodox Medical Treatment
Alternative Medical Treatment
Recovery Time
About the Author
Endnotes
Introduction
If you are an American diabetic, your physician will never tell you that
most cases of diabetes are curable. In fact, if you even mention the "cure"
word around him, he will likely become upset and irrational. His medical
school training only allows him to respond to the word "treatment". For
him, the "cure" word does not exist. Diabetes, in its modern epidemic form,
is a curable disease and has been for at least 40 years. In 2001, the most
recent year for which US figures are posted, 934,550 Americans died from
out-of-control symptoms of this disease.1
Your physician will also never tell you that, at one time, strokes, both
ischaemic and haemorrhagic, heart failure due to neuropathy as well as both
ischaemic and haemorrhagic coronary events, obesity, atherosclerosis,
elevated blood pressure, elevated cholesterol, elevated triglycerides,
impotence, retinopathy, renal failure, liver failure, polycystic ovary
syndrome, elevated blood sugar, systemic candida, impaired carbohydrate
metabolism, poor wound healing, impaired fat metabolism, peripheral
neuropathy as well as many more of today's disgraceful epidemic disorders
were once well understood often to be but symptoms of diabetes.
If you contract diabetes and depend upon orthodox medical treatment, sooner
or later you will experience one or more of its symptoms as the disease
rapidly worsens. It is now common practice to refer to these symptoms as if
they were separable, independent diseases with separate, unrelated
treatments provided by competing medical specialists.
It is true that many of these symptoms can and sometimes do result from
other causes; however, it is also true that this fact has been used to
disguise the causative role of diabetes and to justify expensive,
ineffective treatments for these symptoms.
Epidemic Type II diabetes is curable. By the time you get to the end of
this article, you are going to know that. You're going to know why it isn't
routinely being cured. And, you're going to know how to cure it. You are
also probably going to be angry at what a handful of greedy people have
surreptitiously done to the entire orthodox medical community and to its
trusting patients.
The Diabetes Industry
Today's diabetes industry is a massive community that has grown step by
step from its dubious origins in the early 20th century. In the last 80
years it has become enormously successful at shutting out competitive
voices that attempt to point out the fraud involved in modern diabetes
treatment. It has matured into a religion. And, like all religions, it
depends heavily upon the faith of the believer. So successful has it become
that it verges on blasphemy to suggest that, in most cases, the kindly high
priest with the stethoscope draped prominently around his neck is a
charlatan and a fraud. In the large majority of cases, he has never cured a
single case of diabetes in his entire medical career.
The financial and political influence of this medical community has almost
totally subverted the original intent of our regulatory agencies. They
routinely approve death-dealing, ineffective drugs with insufficient
testing. Former commissioner of the FDA, Dr Herbert Ley, in testimony
before a US Senate hearing, commented: "People think the FDA is protecting
them. It isn't. What the FDA is doing and what the public thinks it's doing
are as different as night and day."2
The financial and political influence of this medical community dominates
our entire medical insurance industry. Although this is beginning to
change, in America it is still difficult to find employer group medical
insurance to cover effective alternative medical treatments. Orthodox
coverage is standard in all states. Alternative medicine is not. For
example, there are only 1,400 licensed naturopaths in 11 states compared to
over 3.4 million orthodox licensees in 50 states.3 Generally, only approved
treatments from licensed, credentialed practitioners are insurable. This,
in effect, neatly creates a special kind of money that can only be spent
within the orthodox medical and drug industry. No other industry in the
world has been able to manage the politics of convincing people to accept
so large a part of their pay in a form that often does not allow them to
spend it as they see fit.
The financial and political influence of this medical community completely
controls virtually every diabetes publication in the country. Many diabetes
publications are subsidised by ads for diabetes supplies. No diabetes
editor is going to allow the truth to be printed in his magazine. This is
why the diabetic only pays about one-quarter to one-third of the cost of
printing the magazine he depends upon for accurate information. The rest is
subsidised by diabetes manufacturers with a vested commercial interest in
preventing diabetics from curing their diabetes. When looking for a
magazine that tells the truth about diabetes, look first to see if it is
full of ads for diabetes supplies.
And then there are the various associations that solicit annual donations
to find a cure for their proprietary disease. Every year they promise that
a cure is just around the corner—just send more money! Some of these very
same associations have been clearly implicated in providing advice that
promotes the progress of diabetes in their trusting supporters. For
example, for years they heavily promoted exchange diets,4 which are in fact
scientifically worthless—as anyone who has ever tried to use them quickly
finds out. They ridiculed the use of glycaemic tables, which are actually
very helpful to the diabetic. They promoted the use of margarine as heart
healthy, long after it was well understood that margarine causes diabetes
and promotes heart failure.5
If people ever wake up to the cure for diabetes that has been suppressed
for 40 years, these associations will soon be out of business. But until
then, they nonetheless continue to need our support.
For 40 years, medical research has consistently shown with increasing
clarity that diabetes is a degenerative disease directly caused by an
engineered food supply that is focused on profit instead of health.
Although the diligent can readily glean this information from a wealth of
medical research literature, it is generally otherwise unavailable.
Certainly this information has been, and remains, largely unavailable in
the medical schools that train our retail doctors.
Prominent among the causative agents in our modern diabetes epidemic are
the engineered fats and oils that are sold in today's supermarkets.
The first step to curing diabetes is to stop believing the lie that the
disease is incurable.
Diabetes History
In 1922, three Canadian Nobel Prize winners, Banting, Best and Macleod,
were successful in saving the life of a fourteen-year-old diabetic girl in
Toronto General Hospital with injectable insulin.6 Eli Lilly was licensed
to manufacture this new wonder drug, and the medical community basked in
the glory of a job well done.
It wasn't until 1933 that rumours about a new rogue form of diabetes
surfaced. This was in a paper presented by Joslyn, Dublin and Marks and
printed in the American Journal of Medical Sciences. This paper, "Studies
on Diabetes Mellitus",7 discussed the emergence of a major epidemic of a
disease which looked very much like the diabetes of the early 1920s, only
it did not respond to the wonder drug, insulin. Even worse, sometimes
insulin treatment killed the patient.
This new disease became known as "insulin-resistant diabetes" because it
had the elevated blood sugar symptom of diabetes but responded poorly to
insulin therapy. Many physicians had considerable success in treating this
disease through diet. A great deal was learned about the relationship
between diet and diabetes in the 1930s and 1940s.
Diabetes, which had a per-capita incidence of 0.0028% at the turn of the
century, had by 1933 zoomed 1,000% in the United States to become a disease
seen by many doctors.8 This disease, under a variety of aliases, was
destined to go on to wreck the health of over half the American population
and incapacitate almost 20% by the 1990s.9
In 1950, the medical community became able to perform serum insulin assays.
These assays quickly revealed that this new disease wasn't classic
diabetes; it was characterised by sufficient, often excessive, blood
insulin levels.
The problem was that the insulin was ineffective; it did not reduce blood
sugar. But since the disease had been known as diabetes for almost 20
years, it was renamed Type II diabetes. This was to distinguish it from the
earlier Type I diabetes, caused by insufficient insulin production by the
pancreas.
Had the dietary insights of the previous 20 years dominated the medical
scene from this point and into the late 1960s, diabetes would have become
widely recognised as curable instead of merely treatable. Instead, in 1950,
a search was launched for another wonder drug to deal with the Type II
diabetes problem.
Cure versus Treatment
This new, ideal, wonder drug would be effective, like insulin, in remitting
obvious adverse symptoms of the disease but not effective in curing the
underlying disease. Thus it would be needed continually for the remaining
life of the patient. It would have to be patentable; that is, it could not
be a natural medication because these are non-patentable. Like insulin, it
would have to be highly profitable to manufacture and distribute. Mandatory
government approvals would be required to stimulate physicians to prescribe
it as a prescription drug. Testing required for these approvals would have
to be enormously expensive to prevent other, unapproved, medications from
becoming competitive.
This is the origin of the classic medical protocol of "treating the
symptoms". By doing this, both the drug company and the doctor could
prosper in business, and the patient, while not being cured of his disease,
was sometimes temporarily relieved of some of his symptoms.
Additionally, natural medications that actually cured disease would have to
be suppressed. The more effective they were, the more they would need to be
suppressed and their proponents jailed as quacks. After all, it wouldn't do
to have some cheap, effective, natural medication cure disease in a
capital-intensive monopoly market specifically designed to treat symptoms
without curing disease.
Often the natural substance really did cure disease. This is why the force
of law has been and is being used to drive the natural, often superior,
medicines from the marketplace, to remove the "cure" word from the medical
vocabulary and to undermine totally the very concept of a free marketplace
in the medical business.
Now it is clear why the "cure" word is so vigorously suppressed by law. The
FDA has extensive Orwellian regulations that prohibit the use of the "cure"
word to describe any competing medicine or natural substance. It is
precisely because many natural substances do actually both cure and prevent
disease that this word has become so frightening to the drug and orthodox
medical community.
The Commercial Value of Symptoms
After the drug development policy was redesigned to focus on ameliorating
symptoms rather than curing disease, it became necessary to reinvent the
way drugs were marketed. This was done in 1949 in the midst of a major
epidemic of insulin-resistant diabetes.
So, in 1949, the US medical community reclassified the symptoms of
diabetes10 along with many other disease symptoms into diseases in their
own right. With this reclassification as the new basis for diagnosis,
competing medical speciality groups quickly seized upon related groups of
symptoms as their own proprietary symptoms set.
Thus the heart specialist, endocrinologist, allergist, kidney specialist
and many others started to treat the symptoms for which they felt
responsible. As the underlying cause of the disease was widely ignored, all
focus on actually curing anything was completely lost.
Heart failure, for example, which had previously been understood often to
be but a symptom of diabetes, now became a disease not directly connected
to diabetes. It became fashionable to think that diabetes "increased
cardiovascular risk". The causal role of a failed blood-sugar control
system in heart failure became obscured.
Consistent with the new medical paradigm, none of the treatments offered by
the heart specialist actually cures, or is even intended to cure, their
proprietary disease. For example, the three-year survival rate for bypass
surgery is almost exactly the same as if no surgery was undertaken.11
Today, over half of the people in America suffer from one or more symptoms
of this disease. In its beginnings, it became well known to physicians as
Type II diabetes, insulin-resistant diabetes, insulin resistance,
adult-onset diabetes or, more rarely, hyperinsulinaemia.
According to the American Heart Association, almost 50% of Americans suffer
from one or more symptoms of this disease. One third of the US population
is morbidly obese; half of the population is overweight. Type II diabetes,
also called adult-onset diabetes, now appears routinely in six-year-old
children.
Many degenerative diseases can be traced to a massive failure of the
endocrine system. This was well known to the physicians of the 1930s as
insulin-resistant diabetes. This basic underlying disorder is known to be a
derangement of the blood-sugar control system by badly engineered fats and
oils. It is exacerbated and complicated by the widespread lack of other
essential nutrition that the body needs to cope with the metabolic
consequences of these poisons.
All fats and oils are not equal. Some are healthy and beneficial; many,
commonly available in the supermarket, are poisonous. The health
distinction is not between saturated and unsaturated, as the fats and oils
industry would have us believe. Many saturated oils and fats are highly
beneficial; many unsaturated oils are highly poisonous. The important
health distinction is between natural and engineered.
There exists great dishonesty in advertising in the fats and oils industry.
It is aimed at creating a market for cheap junk oils such as soy,
cottonseed and rapeseed oils.
With an informed and aware public, these oils would have no market at all,
and the USA—indeed, the world—would have far fewer cases of diabetes.
Epidemiological Lifestyle Link
As early as 1901, efforts had been made to manufacture and sell food
products by the use of automated factory machinery because of the immense
profits that were possible. Most of the early efforts failed because people
were inherently suspicious of food that wasn't farm fresh and because the
technology was poor. As long as people were prosperous, suspicious food
products made little headway. Crisco,12 the artificial shortening, was once
given away free in 21/2 lb cans in an unsuccessful effort to influence
American housewives to trust and buy the product in preference to lard.
Margarine was introduced and was bitterly opposed by the dairy states in
the USA. With the advent of the Depression of the 1930s, margarine, Crisco
and a host of other refined and hydrogenated products began to make
significant penetration into the food markets of America. Support for dairy
opposition to margarine faded during World War II because there wasn't
enough butter for the needs of both the civilian population and the
military.13 At this point, the dairy industry, having lost much support,
simply accepted a diluted market share and concentrated on supplying the
military.
Flax oils and fish oils, which were common in the stores and considered
dietary staples before the American population became diseased, have
disappeared from the shelf. The last supplier of flax oil to the major
distribution chains was Archer Daniels Midland, and it stopped producing
and supplying the product in 1950.
More recently, one of the most important of the remaining, genuinely
beneficial, fats was subjected to a massive media disinformation campaign
that portrayed it as a saturated fat that causes heart failure. As a
result, it has virtually disappeared from the supermarket shelves. Thus was
coconut oil removed from the food chain and replaced with soy oil,
cottonseed oil and rapeseed oil.14 Our parents and grandparents would never
have swapped a fine, healthy oil like coconut oil for these cheap, junk
oils. It was shortly after this successful media blitz that the US populace
lost its war on fat. For many years, coconut oil had been our most
effective dietary weight-control agent.
The history of the engineered adulteration of our once-clean food supply
exactly parallels the rise of the epidemic of diabetes and
hyperinsulinaemia now sweeping the United States as well as much of the
rest of the world.
The second step to a cure for this disease epidemic is to stop believing
the lie that our food supply is safe and nutritious.
The Nature of the Disease
Diabetes is classically diagnosed as a failure of the body to metabolise
carbohydrates properly. Its defining symptom is a high blood-glucose level.
Type I diabetes results from insufficient insulin production by the
pancreas. Type II diabetes results from ineffective insulin. In both types,
the blood-glucose level remains elevated. Neither insufficient insulin nor
ineffective insulin can limit post-prandial (after-eating) blood sugar to
the normal range. In established cases of Type II diabetes, these elevated
blood sugar levels are often preceded and accompanied by chronically
elevated insulin levels and by serious distortions of other endocrine
hormonal markers.
The ineffective insulin is no different from effective insulin. Its
ineffectiveness lies in the failure of the cell population to respond to
it. It is not the result of any biochemical defect in the insulin itself.
Therefore, it is appropriate to note that this is a disease that affects
almost every cell in the 70 trillion or so cells of the body. All of these
cells are dependent upon the food that we eat for the raw materials they
need for self repair and maintenance.
The classification of diabetes as a failure to metabolise carbohydrates is
a traditional classification that originated in the early 19th century when
little was known about metabolic diseases or processes.15 Today, with our
increased knowledge of these processes, it would appear quite appropriate
to define Type II diabetes more fundamentally as a failure of the body to
metabolise fats and oils properly. This failure results in a loss of
effectiveness of insulin and in the consequent failure to metabolise
carbohydrates. Unfortunately, much medical insight into this matter, except
at the research level, remains hampered by its 19th-century legacy.
Thus Type II diabetes and its early hyperinsulinaemic symptoms are
whole-body symptoms of this basic cellular failure to metabolise glucose
properly. Each cell of the body, for reasons which are becoming clearer,
finds itself unable to transport glucose from the bloodstream to its
interior. The glucose then remains in the bloodstream, or is stored as body
fat or as glycogen, or is otherwise disposed of in urine.
It appears that when insulin binds to a cell membrane receptor, it
initiates a complex cascade of biochemical reactions inside the cell. This
causes a class of glucose transporters known as GLUT4 molecules to leave
their parking area inside the cell and travel to the inside surface of the
plasma cell membrane.
When in the membrane, they migrate to special areas of the membrane called
caveolae areas.16 There, by another series of biochemical reactions, they
identify and hook up with glucose molecules and transport them into the
interior of the cell by a process called endocytosis. Within the cell's
interior, this glucose is then burned as fuel by the mitochondria to
produce energy to power cellular activity. Thus these GLUT4 transporters
lower glucose in the bloodstream by transporting it out of the bloodstream
into all the cells of the body.
Many of the molecules involved in these glucose- and insulin-mediated
pathways are lipids; that is, they are fatty acids. A healthy plasma cell
membrane, now known to be an active player in the glucose scenario,
contains a complement of cis-type w=3 unsaturated fatty acids.17 This makes
the membrane relatively fluid and slippery. When these cis- fatty acids are
chronically unavailable because of our diet, trans- fatty acids and short-
and medium-chain saturated fatty acids are substituted in the cell
membrane. These substitutions make the cellular membrane stiffer and more
sticky, and inhibit the glucose transport mechanism.18
Thus, in the absence of sufficient cis omega 3 fatty acids in our diet,
these fatty acid substitutions take place, the mobility of the GLUT4
transporters is diminished, the interior biochemistry of the cell is
changed and glucose remains elevated in the bloodstream.
Elsewhere in the body, the pancreas secretes excess insulin, the liver
manufactures fat from the excess sugar, the adipose cells store excess fat,
the body goes into a high urinary mode, insufficient cellular energy is
available for bodily activity and the entire endocrine system becomes
distorted. Eventually, pancreatic failure occurs, body weight plummets and
a diabetic crisis is precipitated.
Although there remains much work to be done to elucidate fully all of the
steps in all of these pathways, this clearly marks the beginning of a
biochemical explanation for the known epidemiological relationship between
cheap, engineered dietary fats and oils and the onset of Type II diabetes.
Orthodox Medical Treatment
After the diagnosis of diabetes, modern orthodox medical treatment consists
of either oral hypoglycaemic agents or insulin.
• Oral hypoglycaemic agents
In 1955, oral hypoglycaemic drugs were introduced. Currently available oral
hypoglycaemic agents fall into five classifications according to their
biophysical mode of action.19 These classes are: biguanides; glucosidase
inhibitors; meglitinides; sulphonylureas; and thiazolidinediones.
The biguanides lower blood sugar in three ways. They inhibit the normal
release by the liver of its glucose stores, they interfere with intestinal
absorption of glucose from ingested carbohydrates, and they are said to
increase peripheral uptake of glucose.
The glucosidase inhibitors are designed to inhibit the amylase enzymes
produced by the pancreas and which are essential to the digestion of
carbohydrates. The theory is that if the digestion of carbohydrates is
inhibited, the blood sugar level cannot be elevated.
The meglitinides are designed to stimulate the pancreas to produce insulin
in a patient that likely already has an elevated level of insulin in their
bloodstream. Only rarely does the doctor even measure the insulin level.
Indeed, these drugs are frequently prescribed without any knowledge of the
pre-existing insulin level. The fact that an elevated insulin level is
almost as damaging as an elevated glucose level is widely ignored.
The sulphonylureas are another pancreatic stimulant class designed to
stimulate the production of insulin. Serum insulin determinations are
rarely made by the doctor before he prescribes these drugs. They are often
prescribed for Type II diabetics, many of whom already have elevated
ineffective insulin. These drugs are notorious for causing hypoglycaemia as
a side effect.
The thiazolidinediones are famous for causing liver cancer. One of them,
Rezulin, was approved in the USA through devious political infighting, but
failed to get approval in the UK because it was known to cause liver
cancer. The doctor who had responsibility to approve it at the FDA refused
to do so. It was only after he was replaced by a more compliant official
that Rezulin gained approval by the FDA. It went on to kill well over 100
diabetes patients and cripple many others before the fight to get it off
the market was finally won. Rezulin was designed to stimulate the uptake of
glucose from the bloodstream by the peripheral cells and to inhibit the
normal secretion of glucose by the liver. The politics of why this drug
ever came onto market, and then remained in the market for such an
unexplainable length of time with regulatory agency approval, is not
clear.20 As of April 2000, lawsuits commenced to clarify this situation.21
• Insulin
Today, insulin is prescribed for both the Type I and Type II diabetics.
Injectable insulin substitutes for the insulin that the body no longer
produces. Of course, this treatment, while necessary for preserving the
life of the Type I diabetic, is highly questionable when applied to the
Type II diabetic.
It is important to note that neither insulin nor any of these oral
hypoglycaemic agents exerts any curative action whatsoever on any type of
diabetes. None of these medical strategies is designed to normalise the
cellular uptake of glucose by the cells that need it to power their activity.
The prognosis with this orthodox treatment is increasing disability and
early death from heart or kidney failure or the failure of some other vital
organ.
Alternative Medical Treatment
The third step to a cure for this disease is to become informed and to
apply an alternative methodology that is soundly based upon good science.
Effective alternative treatment that directly leads to a cure is available
today for some Type I and for many Type II diabetics. About 5% of the
diabetic population suffers from Type I diabetes; about 95% has Type II
diabetes.22 Gestational diabetes is simply ordinary diabetes contracted by
a woman who is pregnant.
For the Type I diabetic, an alternative methodology for the treatment of
Type I diabetes is now available. It was developed in modern hospitals in
Madras, India, and subjected to rigorous double-blind studies to prove its
efficacy.23 It operates to restore normal pancreatic beta cell function so
that the pancreas can again produce insulin as it should. This approach
apparently was capable of curing Type I diabetes in over 60% of the
patients on whom it was tested. The major complication lies in whether the
antigens that originally led to the autoimmune destruction of these beta
cells have disappeared from or remain in the body. If they remain, a cure
is less likely; if they have disappeared, the cure is more likely. For
reasons already discussed, this methodology is not likely to appear in the
United States any time soon, and certainly not in the American orthodox
medical community.
The goal of any effective alternative program is to repair and restore the
body's own blood-sugar control mechanism. It is the malfunctioning of this
mechanism that, over time, directly causes all of the many debilitating
symptoms that make orthodox treatment so financially rewarding for the
diabetes industry. For Type II diabetes, the steps in the program are:24
• Repair the faulty blood sugar control system. This is done simply by
substituting clean, healthy, beneficial fats and oils in the diet for the
pristine-looking but toxic trans-isomer mix found in attractive plastic
containers on supermarket shelves. Consume only flax oil, fish oil and
occasionally cod liver oil until blood sugar starts to stabilise. Then add
back healthy oils such as butter, coconut oil, olive oil and clean animal
fat. Read labels; refuse to consume cheap junk oils when they appear in
processed food or on restaurant menus. Diabetics are chronically short of
minerals; they need to add a good-quality, broad-spectrum mineral
supplement to the diet.
• Control blood sugar manually during the recovery cycle. Under medical
supervision, gradually discontinue all oral hypoglycaemic agents along with
any additional drugs given to counteract their side effects. Develop
natural blood-sugar control by the use of glycaemic tables, by consuming
frequent small meals (including fibre-rich foods), by regular post-prandial
exercise, and by the complete avoidance of all sugars along with the
judicious use of only non-toxic sweeteners.25 Avoid alcohol until blood
sugar stabilises in the normal range. Keep score by using a pinprick-type
glucose meter. Keep track of everything you do with a medical diary.
• Restore a proper balance of healthy fats and oils when the blood sugar
controller again works. Permanently remove from the diet all cheap, toxic,
junk fats and oils as well as the processed and restaurant foods that
contain them. When the blood sugar controller again starts to work
correctly, gradually introduce additional healthy foods to the diet. Test
the effect of these added foods by monitoring blood sugar levels with the
pinprick-type blood sugar monitor. Be sure to include the results of these
tests in your diary also.
• Continue the program until normal insulin values are also restored after
blood sugar levels begin to stabilise in the normal region. Once blood
sugar levels fall into the normal range, the pancreas will gradually stop
overproducing insulin. This process will typically take a little longer and
can be tested by having your physician send a sample of your blood to a lab
for a serum insulin determination. A good idea is to wait a couple of
months after blood sugar control is restored and then have your physician
check your insulin level. It's nice to have blood sugar in the normal
range; it's even nicer to have this accomplished without excess insulin in
the bloodstream.
• Separately repair the collateral damage done by the disease. Vascular
problems caused by a chronically elevated glucose level will normally
reverse themselves without conscious effort. The effects of retinopathy and
of peripheral neuropathy, for example, will usually self repair. However,
when the fine capillaries in the basement membranes of the kidneys begin to
leak due to chronic high blood glucose, the kidneys compensate by laying
down scar tissue to prevent the leakage. This scar tissue remains even
after the diabetes is cured, and is the reason why the kidney damage is not
believed to self repair.
A word of warning… When retinopathy develops, there may be a temptation to
have the damage repaired by laser surgery. This laser technique stops the
retinal bleeding by creating scar tissue where the leaks have developed.
This scar tissue will prevent normal healing of the fine capillaries in the
eye when the diabetes is reversed. By reversing the diabetes instead of
opting for laser surgery, there is an excellent chance that the eye will
heal completely. However, if laser surgery is done, this healing will
always be complicated by the scar tissue left by the laser.
The arterial and vascular damage done by years of elevated sugar and
insulin and by the proliferation of systemic candida will slowly reverse
due to improved diet. However, it takes many years to clean out the
arteries by this form of oral chelation. Arterial damage can be reversed
much more quickly by using intravenous chelation therapy.26 What would
normally take many years through diet alone can often be done in six months
with intravenous therapy. This is reputed to be effective over 80% of the
time. For obvious reasons, don't expect your doctor to approve of this,
particularly if he's a heart specialist.
Recovery Time
The prognosis is usually swift recovery from the disease and restoration of
normal health and energy levels in a few months to a year or more. The
length of time that it takes to effect a cure depends upon how long the
disease was allowed to develop.
For those who work quickly to reverse the disease after early discovery,
the time is usually a few months or less. For those who have had the
disease for many years, this recovery time may lengthen to a year or more.
Thus, there is good reason to get busy reversing this disease as soon as it
becomes clearly identified.
By the time you get to this point in this article, and if we've done a good
job of explaining our diabetes epidemic, you should know what causes it,
what orthodox medical treatment is all about, and why diabetes has become a
national and international disgrace.
Of even greater importance, you have become acquainted with a self-help
program that has demonstrated great potential to actually cure this disease. 8
About the Author:
Thomas Smith is a reluctant medical investigator, having been forced into
curing his own diabetes because it was obvious that his doctor would not or
could not cure it.
He has published the results of his successful diabetes investigation in
his self-help manual, Insulin: Our Silent Killer, written for the layperson
but also widely valued by the medical practitioner. This manual details the
steps required to reverse Type II diabetes and references the work being
done with Type I diabetes. The book may be purchased from the author at PO
Box 7685, Loveland, Colorado 80537, USA (North American residents send
$US25.00; overseas residents should contact the author for payment and
shipping instructions).
Thomas Smith has also posted a great deal of useful information about
diabetes on his website, http://www.Healingmatters.com He can be
contacted by telephone at +1 (970) 669 9176 and by email at
valley@healingmatters.com
Endnotes:
1. National Center for Health Statistics, "Fast Stats", Deaths/Mortality
Preliminary 2001 data
2. Dr Herbert Ley, in response to a question from Senator Edward Long about
the FDA during US Senate hearings in 1965
3. Eisenberg, David M., MD, "Credentialing complementary and alternative
medical providers", Annals of Internal Medicine 137(12):968 (December 17,
2002)
4. American Diabetes Association and the American Dietetic Association, The
Official Pocket Guide to Diabetic Exchanges, McGraw-Hill/Contemporary
Distributed Products, newly updated March 1, 1998
5. American Heart Association, "How Do I Follow a Healthy Diet?", American
Heart Association
National Center (7272 Greenville Avenue, Dallas, Texas 75231-4596, USA),
http://www.americanheart.org
6. Brown., J.A.C., Pears Medical Encyclopedia Illustrated, 1971, p. 250
7. Joslyn, E.P., Dublin, L.I., Marks, H.H., "Studies on Diabetes Mellitus",
American Journal of Medical Sciences 186:753-773 (1933)
8. "Diabetes Mellitus", Encyclopedia Americana, Library Edition, vol. 9,
1966, pp. 54-56
9. American Heart Association, "Stroke (Brain Attack)", August 28, 1998,
http://www.amhrt.org/ScientificHStats98/05stroke.html;
American Heart Association, "Cardiovascular Disease Statistics", August 28,
1998, http://www.amhrt.org/Heart_and_Stroke_A ... /cvds.html;
"Statistics related to overweight and obesity",
http://niddk.nih.gov/health/nutrit/pubs/statobes.htm ;
http://www.winltdusa.com/about/infocent ... estats.htm
10. "Diabetes Mellitus", Encyclopedia Americana, ibid., pp. 54-55
11. The Veterans Administration Coronary Artery Bypass Co-operative Study
Group, "Eleven-year survival in the Veterans Administration randomized
trial of coronary bypass surgery for stable angina", New Eng. J. Med.
311:1333-1339 (1984); Coronary Artery Surgery Study (CASS), "A randomized
trial of coronary artery bypass surgery: quality of life in patients
randomly assigned to treatment groups", Circulation 68(5):951-960 (1983)
12. Trager, J., The Food Chronology, Henry Holt & Company, New York, 1995
(items listed by date)
13. "Margarine", Encyclopedia Americana, Library Edition, vol. 9, 1966, pp.
279-280
14. Fallon, S., Connolly, P., Enig, M.C., Nourishing Traditions, Promotion
Publishing, 1995;
Enig, M.C., "Coconut: In Support of Good Health in the 21st Century",
http://www.livecoconutoil.com/maryenig.htm
15. Houssay, Bernardo, A., MD, et al., Human Physiology, McGraw-Hill Book
Company, 1955, pp. 400-421
16. Gustavson, J., et al., "Insulin-stimulated glucose uptake involves the
transition of glucose transporters to a caveolae-rich fraction within the
plasma cell membrane: implications for type II diabetes", Mol. Med.
2(3):367-372 (May 1996)
17. Ganong, William F., MD, Review of Medical Physiology, 19th edition,
1999, p. 9, pp. 26-33
18. Pan, D.A. et al., "Skeletal muscle membrane lipid composition is
related to adiposity and insulin action", J. Clin. Invest. 96(6):2802-2808
(December 1995)
19. Physicians' Desk Reference, 53rd edition, 1999
20. Smith, Thomas, Insulin: Our Silent Killer, Thomas Smith, Loveland,
Colorado, revised 2nd
edition, July 2000, p. 20
21. Law Offices of Charles H. Johnson & Associates (telephone 1 800 535
5727, toll free in North America)
22. American Heart Association, "Diabetes Mellitus Statistics",
http://www.amhrt.org
23. Shanmugasundaram, E.R.B. et al. (Dr Ambedkar Institute of Diabetes,
Kilpauk Medical College Hospital, Madras, India), "Possible regeneration of
the Islets of Langerhans in Streptozotocin-diabetic rats given Gymnema
sylvestre leaf extract", J. Ethnopharmacology 30:265-279 (1990);
Shanmugasundaram, E.R.B. et al., "Use of Gemnema sylvestre leaf extract in
the control of blood glucose in insulin-dependent diabetes mellitus", J.
Ethnopharmacology 30:281-294 (1990)
24. Smith, ibid., pp. 97-123
25. Many popular artificial sweeteners on sale in the supermarket are
extremely poisonous and dangerous to the diabetic; indeed, many of them are
worse than the sugar the diabetic is trying to avoid; see, for example,
Smith, ibid., pp. 53-58.
26. Walker, Morton, MD, and Shah, Hitendra, MD, Chelation Therapy, Keats
Publishing, Inc., New Canaan, Connecticut, 1997, ISBN 0-87983-730-6
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